Haloxyfop-R Methyl Ester 10.8% EC

It is a non-selective, connected, and semi-systemic phosphonic acid herbicide inhibition of glutamine synthesis, leading to a complete breakdown of ammonia metabolism and subsequent accumulation of ammonia in tissues. This herbicide also interferes with photosynthesis. Glufosinate ammonium is used to control annual and non-perennial weeds, so this herbicide does not control perennial weeds well. Chlorosis, withering of tissues, and finally, the death of the weed within 1 to 2 weeks are the symptoms of the effect of the herbicide.

It is a selective, systemic post-emergence herbicide. It belongs to the class of cyclohexanediones herbicides and inhibition of acetyl CoA carboxylase (ACCase Inhibitors). Its mode of action is growing points to inhibit the biosynthesis of plant branched-chain fatty acids. This herbicide is absorbed by the aerial parts of the plant.

It is a selective, systemic post-emergence herbicide. It belongs to the class of chloroacetamide herbicides and inhibition of Very Long Chain Fatty Acids (VLCFA). It is taken up readily by the hypocotyls, mesocotyls and coleoptiles, and, to a lesser extent, by the roots of germinating weeds.

It is a selective, systemic post-emergence herbicide. It belongs to the class of sulfonylurea herbicides and inhibition of aceto-lactase synthase (ALS) or Acetohydroxyacid synthase (AHAS) mode of action. Its mode of action is by branched chain amino acid valine, leucine, and isoleucine synthesis inhibition. This herbicide is quickly decomposed by the chemical and biochemical factors of the soil.