Tribenuron methyl 75% DF

It is a selective, systemic post-emergence herbicide. It belongs to the class of Aryloxphenoxy-propionates herbicides and inhibition of acetyl CoA carboxylase (ACCase Inhibitors). Its mode of action is growing points to inhibit the biosynthesis of plant branched-chain fatty acids. This herbicide is absorbed by the aerial parts and roots of the plant.

It is a selective, contact herbicide. It belongs to the class of N-Phenyl-oxadiazolones herbicides and inhibitor of protoporphyrinogen oxidase (PPO). The development of the shoots is stopped as soon as they emerge - their tissues decay very rapidly.

It is a selective, systemic post-emergence herbicide, absorbed by foliage and roots. It belongs to the class of sulfonylurea herbicides and inhibition of aceto-lactase synthase (ALS) or Acetohydroxyacid synthase (AHAS) mode of action. Its mode of action is by branched chain amino acid valine, leucine, and isoleucine synthesis inhibition.

It is a non-selective, connected, and semi-systemic phosphonic acid herbicide inhibition of glutamine synthesis, leading to a complete breakdown of ammonia metabolism and subsequent accumulation of ammonia in tissues. This herbicide also interferes with photosynthesis. Glufosinate ammonium is used to control annual and non-perennial weeds, so this herbicide does not control perennial weeds well. Chlorosis, withering of tissues and finally, the death of the weed within 1 to 2 weeks are the symptoms of the effect of the herbicide.